Toll-like receptor 4 mediates endothelial cell activation through NF-κB but is not associated with endothelial dysfunction in patients with rheumatoid arthritis

Angelo Zoli, Carmine Cardillo, Barbara Tolusso, Gianfranco Ferraccioli, R Menghini, U Campia, M Tesauro, A Marino, V Rovella, G Rodia, F Schinzari, N Di Daniele, M Federici

Risultato della ricerca: Contributo in rivistaArticolo in rivista

27 Citazioni (Scopus)

Abstract

To investigate the effects of TLR4 antagonism on human endothelial cells activation and cytokine expression, and whether the Asp299Gly TLR4 polymorphism is associated with better endothelial function in patients with rheumatoid arthritis (RA).
Lingua originaleEnglish
pagine (da-a)e99053-e99053
RivistaPLoS One
Volume9
DOI
Stato di pubblicazionePubblicato - 2014

Keywords

  • Aorta
  • Arthritis, Rheumatoid
  • Cells, Cultured
  • Disaccharides
  • Endothelium, Vascular
  • Humans
  • Lipopolysaccharides
  • NF-kappa B
  • Phosphorylation
  • Sugar Phosphates
  • Toll-Like Receptor 4

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