TY - JOUR
T1 - Prognostic relevance of MLH1 and MSH2 mutations in hereditary non-polyposis colorectal cancer patients
AU - Russo, A
AU - Sala, P
AU - Alberici, P
AU - Gazzoli, I
AU - Radice, P
AU - Montefusco, C
AU - Torrini, M
AU - Mareni, C
AU - Fornasarig, M
AU - Santarosa, M
AU - Viel, A
AU - Benatti, P
AU - Pedroni, M
AU - De Leon, M
AU - Lucci Cordisco, Emanuela
AU - Genuardi, Maurizio
AU - Messerini, L
AU - Stigliano, V
AU - Cama, A
AU - Curia, M
AU - De Lellis, L
AU - Signoroni, S
AU - Pierotti, M
AU - Bertario, L.
PY - 2009
Y1 - 2009
N2 - Aims and background. Colorectal carcinoma patients from hereditary non-polyposis colorectal cancer families are suggested to have a better prognosis than sporadic colorectal carcinoma cases. Since the majority of hereditary non-polyposis colorectal cancer-related colorectal carcinomas are characterized by microsatellite instability due to germline mutations in DNA mismatch repair genes, this is consistent with the prolonged survival observed in sporadic microsatellite instability-positive colorectal carcinoma compared to microsatellite stable cases. However, a fraction of colorectal carcinoma cases belongs to families that, despite fulfilling the clinical criteria for hereditary non-polyposis colorectal cancer, do not carry mismatch repair gene mutations. Our aim was to verify to what extent the genotypic heterogeneity influences the prognosis of hereditary non-polyposis colorectal cancer patients. Methods. A survival analysis was performed on 526 colorectal carcinoma cases from 204 Amsterdam Criteria-positive hereditary non-polyposis colorectal cancer families. Enrolled cases were classified as MLITH-positive, MSH2-positive and mutation-negative, according to the results of genetic testing in each family. Results. Five-year survival rates were 0.73 (95% Cl, 0.66-0.80), 0.75 (95% CI, 0.66-0.84) and 0.62 (95% Cl, 0.55-0.68) for MLH1-positive, MSH2-positive and mutation-negative groups, respectively (logrank test, P= 0.01). Hazard ratio, computed using Cox regression analysis and adjusted for age, sex, tumor site and stage, was 0.71 (95% Cl, 0.51-0.981 for the mutation-positive compared to the mutation-negative group. Moreover, in the latter group, patients with microsatellite instability-positive colorectal carcinomas showed a better outcome than microsatellite stable cases (5-year survival rates, 0.81 and 0.60, respectively; logrank test, P = 0.006). Conclusions. Our results suggest that the prognosis of hereditary non-polyposis colorectal cancer-related colorectal carcinoma patients depends on the associated constitutional mismatch repair genotype.
AB - Aims and background. Colorectal carcinoma patients from hereditary non-polyposis colorectal cancer families are suggested to have a better prognosis than sporadic colorectal carcinoma cases. Since the majority of hereditary non-polyposis colorectal cancer-related colorectal carcinomas are characterized by microsatellite instability due to germline mutations in DNA mismatch repair genes, this is consistent with the prolonged survival observed in sporadic microsatellite instability-positive colorectal carcinoma compared to microsatellite stable cases. However, a fraction of colorectal carcinoma cases belongs to families that, despite fulfilling the clinical criteria for hereditary non-polyposis colorectal cancer, do not carry mismatch repair gene mutations. Our aim was to verify to what extent the genotypic heterogeneity influences the prognosis of hereditary non-polyposis colorectal cancer patients. Methods. A survival analysis was performed on 526 colorectal carcinoma cases from 204 Amsterdam Criteria-positive hereditary non-polyposis colorectal cancer families. Enrolled cases were classified as MLITH-positive, MSH2-positive and mutation-negative, according to the results of genetic testing in each family. Results. Five-year survival rates were 0.73 (95% Cl, 0.66-0.80), 0.75 (95% CI, 0.66-0.84) and 0.62 (95% Cl, 0.55-0.68) for MLH1-positive, MSH2-positive and mutation-negative groups, respectively (logrank test, P= 0.01). Hazard ratio, computed using Cox regression analysis and adjusted for age, sex, tumor site and stage, was 0.71 (95% Cl, 0.51-0.981 for the mutation-positive compared to the mutation-negative group. Moreover, in the latter group, patients with microsatellite instability-positive colorectal carcinomas showed a better outcome than microsatellite stable cases (5-year survival rates, 0.81 and 0.60, respectively; logrank test, P = 0.006). Conclusions. Our results suggest that the prognosis of hereditary non-polyposis colorectal cancer-related colorectal carcinoma patients depends on the associated constitutional mismatch repair genotype.
KW - ADJUVANT CHEMOTHERAPY
KW - COLON-CANCER
KW - CRITERIA
KW - FAMILIES
KW - FEATURES
KW - HNPCC
KW - LYNCH-SYNDROME
KW - MICROSATELLITE-INSTABILITY STATUS
KW - SURVIVAL
KW - TUMOR-INFILTRATING LYMPHOCYTES
KW - ADJUVANT CHEMOTHERAPY
KW - COLON-CANCER
KW - CRITERIA
KW - FAMILIES
KW - FEATURES
KW - HNPCC
KW - LYNCH-SYNDROME
KW - MICROSATELLITE-INSTABILITY STATUS
KW - SURVIVAL
KW - TUMOR-INFILTRATING LYMPHOCYTES
UR - http://hdl.handle.net/10807/57720
U2 - 10.1177/030089160909500616
DO - 10.1177/030089160909500616
M3 - Article
SN - 0300-8916
VL - 95
SP - 731
EP - 738
JO - Tumori
JF - Tumori
ER -