Impact of Chronic Kidney Disease and Platelet Reactivity on Clinical Outcomes Following Percutaneous Coronary Intervention

F. Mangiacapra*, A. Sticchi, E. Bressi, R. Mangiacapra, M. M. Viscusi, I. Colaiori, E. Ricottini, I. Cavallari, S. Spoto, G. P. Ussia, Pietro Manuel Ferraro, F. Grigioni

*Autore corrispondente per questo lavoro

Risultato della ricerca: Contributo in rivistaArticolo

Abstract

We investigated the interaction between chronic kidney disease (CKD) and high platelet reactivity (HPR) in determining long-term clinical outcomes following elective PCI for stable coronary artery disease (CAD). A total of 500 patients treated with aspirin and clopidogrel were divided based on the presence of CKD (defined as glomerular filtration rate of < 60 ml/min/1.73 m2) and HPR (defined as a P2Y12 reaction unit value ≥ 240 at VerifyNow assay). Primary endpoint was the occurrence of major adverse clinical events (MACE) at 5 years. Patients with both CKD and HPR showed the highest estimates of MACE (25.6%, p = 0.005), all-cause death (17.9%, p = 0.004), and cardiac death (7.7%, p = 0.004). The combination of CKD and HPR was an independent predictor of MACE (HR 3.12, 95% CI 1.46–6.68, p = 0.003). In conclusion, the combination of CKD and HPR identifies a cohort of patients with the highest risk of MACE at 5 years.
Lingua originaleInglese
pagine (da-a)N/A-N/A
RivistaJournal of Cardiovascular Translational Research
Numero di pubblicazioneN/A
DOI
Stato di pubblicazionePubblicato - 2021

All Science Journal Classification (ASJC) codes

  • Medicina Molecolare
  • Genetica
  • Scienze Farmaceutiche
  • Cardiologia e Medicina Cardiovascolare
  • Genetica (clinica)

Keywords

  • Chronic kidney disease
  • Coronary artery disease
  • Percutaneous coronary intervention
  • Platelet reactivity

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